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0883073808314157v1
23/7/810    most recent
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First published on May 12, 2008, doi:10.1177/0883073808314157

Journal of Child Neurology 2008;23:810.

A more recent version of this article appeared on July 1, 2008

Article

Pleural Effusion: Beta-Trace Protein in Diagnosing Ventriculoperitoneal Shunt Complications

Mark Born, MD1*, Stefanie Reichling, M.D.2, and Jörg Schirrmeister, M.D.2

1 Department of Radiology–Pediatric Radiology
2 Department of Pediatrics, University of Bonn, Bonn, Germany

* To whom correspondence should be addressed. E-mail: Mark.Born{at}ukb.uni-bonn.de.


  Abstract
Catheter dysfunction is a common complication with ventriculoperitoneal shunts. Apart from infection, obstruction, and leakage, migration of the shunt tip may cause particular problems. Pleural effusion is easily classified as a shunt complication if a transdiaphragmatic migration of a shunt can be demonstrated. If, however, the tip of the shunt is found adjacent to the diaphragm, it is difficult to decide if the effusion is caused by the ventriculoperitoneal shunt. Different diagnostic methods can be used in this situation. Below we report a case of pleural effusion—without shunt migration—which was revealed to be a shunt complication by quantifying beta-trace protein in the effusion.


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