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Journal of Child Neurology
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Differential Involvement of the Brain in Neonatal Asphyxia: A Pathogenic Explanation

Raj D. Sheth, MD

Department of Neurology

John B. Bodensteiner, MD

Department of Pediatrics

Jack E. Riggs, MD

Department of Medicine

Sydney S. Schochet, JR, MD

Department of Pathology, West Virginia University Health Sciences Center, Morgantown, WV

Multiple cystic lesions in brain parenchyma supplied by the anterior cerebral circulation is a recognized pattern of cerebral injury associated with hypoxic-ischemic encephalopathy in the term infant. This report presents a series of seven infants (gestational age, 39.3 ± 2.8 weeks; range, 36 to 44 weeks) who developed multicystic encephalomalacia in the distribution of the anterior cerebral circulation after severe neonatal asphyxia. Cerebral imaging and pathologic studies demonstrate relative preservation of the cerebellum, brain stem, and cerebral structures supplied by the vertebrobasilar circulation. Compared to the vertebrobasilar vasculature, the anterior cerebral vessels in the term infant have dense sympathetic innervation. Asphyxia, a potent sympathetic stimulator, may induce vasoconstriction in the anterior circulation and differentially accentuate the effects of hypoxia/ischemia on cerebral tissue. (J Child Neurol 1995;10:464-466).

Journal of Child Neurology, Vol. 10, No. 6, 464-466 (1995)
DOI: 10.1177/088307389501000608
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This article has been cited by other articles:


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 J Child NeurolHome page
A. I. Maller, L. L. Hankins, J. W. Yeakley, and I. J. Butler
Rolandic Type Cerebral Palsy in Children as a Pattern of Hypoxic-Ischemic Injury in the Full-Term Neonate
J Child Neurol, July 1, 1998; 13(7): 313 - 321.
[Abstract] [PDF]

Home page
 J Child NeurolHome page
R. D. Sheth and J. B. Bodensteiner
Hypertensive Encephalopathy and Sympathetic Innervation of the Cerebral Circulation: A Comment
J Child Neurol, July 1, 1996; 11(4): 347 - 347.
[PDF]


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