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Journal of Child Neurology
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Hypopotassemic Paralysis: A Rare Presentation of Proximal Renal Tubular Acidosis

Gülhis Deda, MD

Department of Pediatric Neurology

Mesiha Ekim, MD

Department of Pediatric Nephrology

Alev Gfven, MD

Department of Pediatric Neurology

Ugur Karagöl, MD

Department of Pediatric Neurology

Necmiye Tümer, MD

Department of Pediatric Nephrology Ankara University Faculty of Medicine Ankara, Turkey

Neurologic manifestations can accompany systemic diseases, and primary disease can be identified with a careful history, physical examination, and laboratory investigations. A 14-year-old girl with paraplegia and absence of deep tendon reflexes in the lower extremities after 2 days of vomiting and diarrhea was referred to our pediatric neurology department with a diagnosis of Guillain-Barré syndrome. Short stature, dehydration, motor and mental retardation, bilateral cataracts, glaucoma, and band keratopathy were detected on physical examination. Hypopotassemia and severe metabolic acidosis were found on biochemical examination. Her paraplegia improved after appropriate fluid and electrolyte replacement, but metabolic acidosis persisted after cessation of intravenous therapy, and isolated proximal renal tubular acidosis was detected. Because she had isolated proximal renal tubular acidosis and other abnormalities, she was diagnosed with Donckerwolcke-Winsnes syndrome. (J Child Neurol 2001;16:770-771).

Journal of Child Neurology, Vol. 16, No. 10, 770-771 (2001)
DOI: 10.1177/088307380101601013
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S. Horita, H. Yamada, J. Inatomi, N. Moriyama, T. Sekine, T. Igarashi, Y. Endo, M. Dasouki, M. Ekim, L. Al-Gazali, et al.
Functional Analysis of NBC1 Mutants Associated with Proximal Renal Tubular Acidosis and Ocular Abnormalities
J. Am. Soc. Nephrol., August 1, 2005; 16(8): 2270 - 2278.
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