Advanced Search

Journal Navigation

Journal Home

Subscriptions

Archive

Contact Us

Table of Contents

Click here for more information

Click here to sign up for SAGE Journal Email Alerts today!

Sign In to gain access to subscriptions and/or personal tools.
Journal of Child Neurology
This Article
Right arrow Full Text (PDF)
Right arrow References
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to Saved Citations
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Request Reprints
Right arrow Add to My Marked Citations
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Right arrow Citing Articles via Scopus
Google Scholar
Right arrow Articles by Marks, W. A.
Right arrow Articles by Bodensteiner, J. B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Marks, W. A.
Right arrow Articles by Bodensteiner, J. B.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us   Add to Digg   Add to Reddit   Add to Technorati  
What's this?

HLA Patterns in Children With Parainflammatory Leukoencephalomyelitis

Warren A. Marks, MD

Fort Worth Child Neurology Associates, Fort Worth, TX

Robert M. Shuman, MD

Department of Neurology, University of Oklahoma Health Sciences Center, Oklahoma City, OK

John B. Bodensteiner, MD

Department of Neurology, West Virginia University, Morgantown, WV

We have evaluated the human leukocyte antigen (HLA) phenotype of six children with parainflammatory leukoencephalomyelitis (PIL). Patients with PIL demonstrate an increased prevalence of the HLA-A1, -A28, -B44, -DR6, and -DR7 antigens. These HLA associations are different from those reported in other inflammatory demyelinating diseases, including multiple sclerosis, optic neuritis, Guillain-Barré syndrome, and chronic relapsing inflammatory polyneuropathy. The HLA constitution of the patient appears to be one important host factor in determining the nature of the immune response to an encephalitogenic challenge. (J Child Neurol 1991;6:150-154).

Journal of Child Neurology, Vol. 6, No. 2, 150-154 (1991)
DOI: 10.1177/088307389100600211


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati    What's this?


This article has been cited by other articles:


Home page
J Child NeurolHome page
M. Kleiman and P. Brunquell
Acute Disseminated Encephalomyelitis: Response to Intravenous Immunoglobulin?
J Child Neurol, November 1, 1995; 10(6): 481 - 483.
[PDF]